Ar Ca 2+ increase under Ca 2+ -free physiological buffer (Figure 3E). These results indicated that the intracellular Ca2+ increase by LTA may come from the intracellularWang et al. Journal of Neuroinflammation 2010, 7:84 http://www.jneuroinflammation.com/content/7/1/Page 7 ofFigure 2 LTA-induced c-Jun phosphorylation is mediated through JNK in RBA-1. (A) Cells were pretreated with SP600125 (1 M) f
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